Last edited by Kajizil
Thursday, May 14, 2020 | History

4 edition of Life-threatening arrhythmias during ischemia and infarction found in the catalog.

Life-threatening arrhythmias during ischemia and infarction

  • 87 Want to read
  • 14 Currently reading

Published by Raven Press in New York .
Written in English

    Subjects:
  • Cardiac arrest.,
  • Arrhythmia.,
  • Myocardial infarction -- Complications.

  • Edition Notes

    Includes bibliographies and index.

    Statementeditors, David J. Hearse, Allan S. Manning, Michiel J. Janse.
    ContributionsHearse, David J., Manning, Allan S., Janse, Michiel Johannes.
    Classifications
    LC ClassificationsRC685.C173 L54 1987
    The Physical Object
    Paginationx, 229 p. :
    Number of Pages229
    ID Numbers
    Open LibraryOL2739805M
    ISBN 100881672661
    LC Control Number86042585

    A comparison of electrocardiographic changes during reperfusion of acute myocardial infarction by thrombolysis or percutaneous transluminal coronary angioplasty. Am Heart J ; Hackett D, McKenna W, Davies G, Maseri A. Reperfusion arrhythmias are rare during acute myocardial infarction and thrombolysis in man. Patients are screened for significant arrhythmias and other possibly significant ECG-patterns directly after discharge and two weeks after myocardial infarction using wearable devices. The home monitoring data will be linked with extensive data from electronic health records collected before, during hospital stay and after discharge.

    It is known that myocardial ischaemia and infarction leads to severe. metabolic and electrophysiological changes that induce silent or symptomatic life-threatening arrhythmias. Sudden cardiac death is most often attributed to this pathophysiology, but many patients sur-vive the early stage of an acute coronary syndrome (ACS) reachingCited by:   Flecainide increased mortality following myocardial infarction (MI) in the Cardiac Arrhythmias Suppression (CAST) trial (Echt et al., ) in which patients surviving acute MI (and who are characteristically at high risk of further acute MI) were randomized to receive maintenance antiarrhythmic therapy or by:

      92 Sinus Bradycardia Physiological variant due to strong vagal tone or atheletic training. Rate as low as 50 at rest and 40 during sleep. Common causes: Hypothermia, hypothyroidism, Drug therapy with betablockers, digitalis and other antiarrhythmic drugs. Acute ischemia and infarction of the sinus node (as a complication of acute myocardial. Arrhythmias and arrhythmology. 24 Chapters. Mechanisms of cardiac arrhythmias: from automaticity to re-entry (reentry) Aberrant ventricular conduction (aberrancy, aberration) Myocardial Ischemia & Infarction. 20 Chapters Conduction Defects. 11 Chapters. Cardiac Hypertrophy & Enlargement. 5 Chapters.


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Life-threatening arrhythmias during ischemia and infarction Download PDF EPUB FB2

Life-Threatening Arrhythmias During Ischemia and Infarction: Medicine & Health Science Books @ ed by: `This book will provide the cardiologist valuable information on the various mechanisms of lethal arrhythmias arising from myocardial ischemia and infarction.' Acta Price: $ Life-threatening arrhythmias during ischemia and infarction.

New York: Raven Press, © (OCoLC) Online version: Life-threatening arrhythmias during ischemia and infarction. New York: Raven Press, © (OCoLC) Document Type: Book: All Authors / Contributors: David J Hearse; Allan S Manning; Michiel Johannes Janse.

Lethal Arrhythmias Resulting from Myocardial Ischemia and Infarction Proceedings of the Second Rappaport Symposium. Editors: Rosen, Michael R., Palti, Yoram (Eds.) Free Preview.

Silent ischemic events were associated with the initiation of life-threatening ventricular arrhythmias in five patients with induced or spontaneous focal coronary artery spasm (or both).Cited by: How the head rules the heart, in Life Threatening Arrhythmias During Ischemia and Infarction, D.J.

Hearse, A.S. Manning, and M Arrhythmias in the Early Ischemic Period. In: Rosen M.R., Palti Y. (eds) Lethal Arrhythmias Resulting from Myocardial Ischemia and Infarction.

Developments in Cardiovascular Medicine, vol Springer, Boston, by: 1. These results suggest that a subclinical mutation in SCN5A resulting in a loss of function may predispose to life-threatening arrhythmias during acute ischemia. In another cohort of patients who developed long QT intervals and Torsade de Pointes (TdP) arrhythmias in days 2–11 following an AMI, a genetic screen of all long QT genes was by: Journals & Books; Register Sign in.

The intention of this review has been to summarise the current state of knowledge regarding the arrhythmias induced by myocardial ischaemia and infarction. Both clinical and experimental aspects were considered. There has been some progress toward understanding the electrophysiological mechanisms Cited by: Ischemia and Myocardial Infarction.

Type A acute aortic dissection is a life-threatening vascular emergency because of its high morbidity and mortality. hypoxemia, myocardial ischemia. Significant ST-segment depression can occur during AVNRT, and represents repolarization changes rather than myocardial ischemia.

Treating the rhythm needs to be divided into acute therapy and prevention of recurrence. As with all arrhythmias causing hemodynamic compromise, electrical cardioversion would be appropriate. Supraventricular or ventricular arrhythmias may be present, either as precipitating factor or as a result of ischemia.

It's important to detect signs of diseases that may contribute to or accompany atherosclerotic heart disease, eg DM, xanthelasma, tendinous xanthomas, HTN, thyrotoxicosis, myxedema, or peripheral artery disease.

It can create a life-threatening situation, and the vascular diseases related to ischemia are among the first causes of mortality and morbidity in the world.

arrhythmias resulting from. Acute-myocardial infarction (AMI)-induced ventricular fibrillation (VF) frequently occurs without warning, often leading to death within minutes in patients who do not receive prompt medical attention.

Identification of patients at risk of AMI-induced lethal ventricular arrhythmias remains an unmet medical by: Rationale of Therapy in the Patient with Acute Myocardial Infarction and Life-Threatening Arrhythmias: A Focus on Bretylium BENEDICT R.

LUCCHESI, PhD, MD Experimental evidence suggests a number of pathologic and electrophysiologic mechanisms that may help initiate ventricular arrhythmias accom- panying myocardial ischemia and by: 7. Ventricular arrhythmias during the acute phase of myocardial infarction are common and account for approximately 80 % of sudden cardiac death cases.

A biphasic curve has been observed in various species and possibly applies also in by: 3. The cellular and electrophysiological changes that accompany infarct healing begin early, but can continue for many months, and patients who have suffered a myocardial infarction remain susceptible to ventricular arrhythmia for life.

Early during infarct healing the transmembrane potential, action potential amplitude and duration, and upstroke velocity are moderately reduced, but not to the extent seen during acute ischemia Cited by: 4.

The effect of antiarrhythmic drugs on life-threatening arrhythmias induced by the interaction between acute myocardial ischemia and sympathetic hyperactivity Transient myocardial ischemia, with attendant sympathetic hyperactivity, seems to play a major role in sudden cardiac death among patients with ischemic heart by:   The effect of antiarrhythmic drugs on life-threatening arrhythmias induced by the interaction between acute myocardial ischemia and sympathetic hyperactivity.

Heart J. – ().Cited by: Life threatening arrhythmias in intracranial hemorrhage Electro- cardiographic changes simulating myocardial ischemia and infarction associated with spontaneous intra- cranial hemorrhage.

Czrculatmn Manning GWElectrocardiographic changes simulating myocardial ischemia and infarction associated with spontaneous Author: Howard S. Schwartz, Philip R. Yarnell. We obtained the recordings promptly, with patients attached to the monitor at a mean time of 7 (median 4) hours from the onset of symptoms, and within 49 (median 44) minutes of arrival to the ED.

Because PVCs commonly develop during ischemia, it would follow that arrhythmias would be more likely to occur early in the patient’s clinical course. Specifically, reperfusion after a brief period of myocardial ischemia can lead to lethal arrhythmias (Tennant and Wiggers ; Wit and Janse ).

In this context, Prinzmetal et al. () first described a variant form of resting, ST segment elevation angina caused by coronary artery by: 5. The occurrence of life-threatening arrhythmias consisting of VT, VF, asystole, and complete AV block was determined during the hospital stay.

VTs lasting >30 s were classified as sustained. Mortality 1 year after initial presentation was obtained by telephone contact with the patient or the treating physician and compared between patients Cited by: challenge) may be required to establish the diagnosis. Arrhythmias, both ventricular tachyarrhythmias and heart block, can occur during the ischaemic episode.

Cardiac syndrome X refers to those patients with a good history of angina, a positive exercise test and angiographically normal coronary arteries. They.